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TSAI LABORATORY

PRINCIPAL INVESTIGATOR

Jonathan

Jonathan Tsai, MD, PhD

Jonathan received his MD and PhD in Developmental Biology from Stanford in 2018, where he worked on lineage tracing tissue specific progenitor cells in the lab of Dr. Irving Weissman. He completed his residency in Clinical Pathology and fellowship in Molecular Genetic Pathology at the Brigham and Women’s Hospital. He completed his post-doctoral fellowship in the laboratory of Dr. Benjamin Ebert. Jonathan is currently an Instructor of Pathology at Harvard Medical School and an Associate Pathologist at the Brigham and Women’s Hospital. His research focuses on understanding the contribution of ubiquitination and protein degradation on transcription in multiple cancer models.

POSTDOCTORAL FELLOWS

Dan

Dan Hou, PhD

Dan received her PhD from the University of Chinese Academy of Sciences and joined the Tsai Lab in August of 2024. By utilizing molecular biology, cell biology, and biochemistry techniques, she is exploring the regulatory mechanisms of epigenetic factors and post-translational modifications in human diseases and will apply this knowledge to advance the understanding and treatment of human diseases.

RESEARCH ASSISTANTS

Haniya

Haniya Sperling, BA

Haniya graduated from the University of Massachusetts, Amherst with a BA in Philosophy and a minor in Biology. She joined the Tsai Lab in July of 2024.

UNDERGARDUATE STUDENTS

Marwa

Marwa Abdel-Hak

Marwa is a rising sophomore at Harvard College concentrating in Human Developmental and Regenerative Biology on a pre-med track. She has a passion for medicine and public health and many of my interests are at the intersection of the two. Marwa is excited to contribute to research in the Tsai Lab, especially as our work can have major implications for hormone-driven diseases!

AFFILIATES

Justine

Justine Rutter, BS

Justine graduated from Duke University with a BS in Biology. She is an MD/PhD student at Harvard in the laboratory of Dr. Benjamin Ebert and works closely with the Tsai Lab on hormone receptor degradation.

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